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A 13-year-old boy was the victim of attempted strangulation.His condition had returned to normal by the sixth day after the assault;however,from the seventh day,choreoathetosis,dystonia,and marked pseudobulbar paralysis developed in the boy.The computed tomographic scans and T2-weighted magnetic resonance images that were obtained at this time revealed low- density and high-signal intensities in the region of the bilateral putamen and caudate nucleus.These symptoms and the changes in his computed tomographic scans and magnetic resonance images subsided gradually during a 2-month period.Sequential analysis of the cerebrospinal fluid for y- aminobutyric acid and dopamine concentrations during his illness revealed reciprocal changes with normal recovery.Because of the delayed onset of neurological changes and the cerebrospinal fluid showing reversible symptoms,the delayed encephalopathy after strangulation had been related to the biochemical alterations that followed anoxia in the vulnerable basal ganglia. |
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basal ganglia,lesion of
basal ganglia,lesion,bilateral
carotid artery occlusion,neck-external and internal art patency with
CAT scan
CAT scan,abnormal
caudate nucleus,lesion of,bilateral
encephalopathy
encephalopathy,anoxic
encephalopathy,post anoxic
hypoxia
hypoxic encephalopathy
MRI
MRI,abnormal
putamen,lesion of
putamen,lesion of,bilateral
strangulation
striatum,lesion of
striatum,lesion of,bilateral
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